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Mouse NGAL ELISA Kit
Catalog #
E-90NGL
Product Size
1.0
plate
The Mouse NGAL ELISA Kit (E-90NGL) from ICL is a sensitive and reliable assay for the quantitative measurement of Neutrophil Gelatinase‑Associated Lipocalin (NGAL / Lipocalin-2) in mouse plasma, serum, or urine — ideal for research on kidney injury, renal disease, or related pathologies.
Target & specificity: Detects mouse NGAL (Lipocalin-2) with a goat-host antibody recognizing the protein.
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Performance: Detection range from 0.125 ng/mL to 4.0 ng/mL, with a sensitivity down to 0.063 ng/mL.
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Sample types: Supports plasma, serum or urine samples.
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Assay time: Fast — complete readout in ~80 minutes.
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Use case: Enables researchers to monitor NGAL levels as an early biomarker for renal injury, nephrotoxicity, or other kidney-related pathologies in mouse models.
Because NGAL (Lipocalin-2) is rapidly upregulated and secreted by injured kidney tubular cells, this ELISA kit offers a powerful tool for preclinical studies of acute kidney injury (AKI), chronic kidney disease, drug-induced nephrotoxicity, or any condition involving renal damage or stress.
| Antibody Type | ELISA |
|---|---|
| Host | Goat |
| Target Species | Mouse |
| Specificity/Target | NGAL (Lipocalin-2) |
| Size | 1.0 |
| Detection Range | 0.125 ng/ml - 4 ng/ml |
| Sensitivity | 0.063 ng/ml |
| Assay Time | 80 min. |
| Sample Type | Plasma, Serum, Urine |
| Storage | 2-8C |
Data Sheet
SDS
Publications citing the use of ICL's Mouse NGAL ELISA Kit:
Jang HR et al. Aging has small effects on initial ischemic acute kidney injury development despite changing intrarenal immunologic micromilieu in mice Am J Physiol Renal Physiol 310: F272–F283, 2016.
Sohn M, Kim K, Uddin MJ, et al. Delayed treatment with fenofibrate protects against high-fat diet-induced kidney injury in mice: the possible role of AMPK autophagy. Am J Physiol Renal Physiol. 2017;312(2):F323-F334. doi:10.1152/ajprenal.00596.2015
Jimenez RCA et al. Fructokinase activity mediates dehydration-induced renal injury. Kidney International. 2014;86(2):294-302. doi:10.1038/ki.2013.492.
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